Proteoglycans in Lung DiseaseCRC Press, 16 oct 2002 - 480 páginas Discusses new treatment strategies for malignant mesothelioma, pulmonary edema, fibrosis, asthma, emphysema, and bronchiectasis. |
Índice
Chapter 1 Proteoglycans of the Lung | 1 |
Chapter 2 Hyaluronan in Lung Function | 23 |
Chapter 3 Morphological Tools for Studying Lung Proteoglycans | 37 |
Chapter 4 Proteoglycans Lung Physiology and Mechanical Strain | 55 |
Chapter 5 Hyaluronan and Its Receptors in Lung Health and Disease | 73 |
Chapter 6 Hyaluronan and Hyaladherin Signaling in the Lung | 107 |
Chapter 7 Hyaluronan in Malignant Mesothelioma | 135 |
Chapter 8 Matrix Proteoglycans in Development of Pulmonary Edema | 143 |
Chapter 12 Role of Proteoglycans in the Development and Pathogenesis of Emphysema | 241 |
Chapter 13 Proteoglycans in Normal and Pathological Bronchial Mucus | 269 |
Chapter 14 Vascular Proteoglycans | 291 |
Chapter 15 Degradation of Lung Matrix Proteoglycans in Bronchiectasis | 323 |
Chapter 16 Effect of Hypoxia on Glycosaminoglycan Synthesis by Lung Cells | 335 |
Chapter 17 Integrins Proteoglycans and Lung Disease | 351 |
Chapter 18 Heparin as a Potential Therapeutic Agent to Reverse Vascular Remodeling | 377 |
AUTHOR INDEX | 399 |
Chapter 9 The Role of Small Proteoglycans in the Formation of Fibrosis | 169 |
Chapter 10 Versican in the Cell Biology of Pulmonary Fibrosis | 191 |
Chapter 11 Decorin in Asthma | 213 |
| 455 | |
BACK COVER | 460 |
Otras ediciones - Ver todo
Términos y frases comunes
activity adhesion airway alterations alveolar associated asthma biglycan binding Biochem Biol Chem bronchial Cell Biol Cell Mol cells changes characterized chondroitin chronic Clin collagen components containing core protein culture cytokines cytoplasmic decorin degradation disease domain edema edited effect emphysema endothelial cells expression extracellular matrix fibroblasts fibronectin fibrosis Figure fluid function gene glycosaminoglycan groups growth factor heparan sulfate heparan sulfate proteoglycan heparin human hyaluronan hyaluronic acid important increased induced inflammation inhibition injury integrin interaction interstitial Invest involved lesions lung lung tissue macrophages mechanical mediated migration molecular molecules neutrophils normal patients Physiol platelet-derived growth factor present production proliferation properties protein pulmonary receptor region regulation Respir response RHAMM role secretion shown signaling smooth muscle cells specific stimulation structure studies suggest surface synthesis tion tissue transforming growth vascular versican vitro wall wound