Focus on Leukemia ResearchNova Publishers, 2004 - 190 páginas This new book brings together leading research from around the globe. Leukaemia is cancer that begins in blood cells. In people with leukaemia, the bone marrow produces abnormal white blood cells. The abnormal cells are leukaemia cells. At first, leukaemia cells function almost normally. In time, they may crowd out normal white blood cells, red blood cells, and platelets. The scope of the book includes the four common types of leukaemia: chronic lymphocytic leukaemia (chronic lymphoblastic leukaemia, CLL) - most often affecting people over age 55; chronic myeloid leukaemia (chronic myelogenous leukaemia, CML) - affects mainly adults; acute lymphocytic leukaemia (acute lymphoblastic leukaemia, ALL) - the most common type of leukaemia in young children; and acute myeloid leukaemia (acute myelogenous leukaemia, AML) - which occurs in both adults and children. New advances in diagnosis, pathogenesis and therapy are also presented. |
Índice
1 | |
The Immune Response to Chronic Myeloid Leukaemia | 27 |
Pathogenesis of Testicular Acute Lymphoblastic Leukaemia | 61 |
Acute Tumor Lysis Syndrome Induced by Corticosteroids | 83 |
Paraffin Section Immunotyping of Leukaemias | 95 |
Cellular Immunotherapy of Leukemia | 113 |
Therapeutic Activity of Cladribine in Patients with Bcell Chronic Lymphocytic Leukemia | 139 |
Biology Natural History and Clinical Features of TELAML 1Positive Childhood Leukaemias | 163 |
185 | |
Términos y frases comunes
acid activity acute lymphoblastic leukemia acute myeloid allogeneic antibodies apoptosis ATLS autologous B-CLL BCR-ABL fusion binding blasts Blood bone marrow breakpoint Cancer CD4+ T-cell CD8+ cell lines cell responses chemotherapy childhood acute lymphoblastic chlorambucil chromosome chronic lymphocytic leukemia chronic myelogenous leukemia chronic myeloid leukemia cladribine Clin clinical CML cells CML patients cytogenetic cytokines cytotoxic T lymphocytes dendritic cells detected diagnosis domain donors dose effect epitopes expression function fusion gene fusion peptide fusion protein Haematol hematological hematopoietic HLA class human imatinib immune response immunogenic immunotherapy induced Inhibin inhibit Jahnukainen junctional peptides leukaemic cells leukemia cells lymphoma malignancies minimal residual disease molecular molecules normal oncogene patients with chronic peptide prednisone proteinase receptor remission reported Robak specific stem cell survival target TEL/AML1 fusion TEL/AML1-positive testicular relapse testis therapy tissue translocation transplantation treatment tumor cells tumor lysis syndrome tyrosine kinase vitro vivo
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